Harvard
University educated archaeologist and president of the Paleontological Research
Corporation, Dr. Joel Klenck, states an array of archaeological discoveries
evidence a crisis during the reign of the Egyptian Pharaoh Thutmose II (ca.
1,492-1,479 B.C.) in the Eighteenth Dynasty.
An inscription
by the succeeding Pharaoh Hatshepsut (ca. 1,479-1,457 B.C.) in her Underground
Temple at Speos Artemidos states that Egypt was “ruined” and “had gone to
pieces” before the beginning of her reign. Hatshepsut’s inscription also states
that a population of “vagabonds” emerged from former Asiatic populations that
once controlled northern Egypt and caused this ruination. Hatshepsut notes these
vagabonds were responsible for “overthrowing that which had been
made”.
Klenck comments,
“The reign of Thutmose II ended between 79 and 86 years after Seqenenre Tao II
(ca. 1,560-1,555 B.C.) began to reconquer northern Egypt from foreign Hyksos
populations, who controlled Egypt during the Second Intermediate Period (ca.
1,650-1,550 B.C.). Egyptian texts are clear that the son of Tao II, Ahmose I,
conquered the Hyksos and captured their capital at Avaris around 1,550 B.C. Yet,
this inscription by Hatshepsut notes another population remained in Egypt from
‘the midst’ of the ‘Asiatics’ and ruined Egypt ‘down to my majesty’ or before
the beginning of her reign.”
Further, there is
evidence that disease affected the royal court before the reign of Hatshepsut.
The mummy of Thutmose II is the only corpse of a pharaoh during the Eighteenth
Dynasty covered with cysts from an unknown malady. These lesions coat the back,
waist, arms and legs of Thutmose II and exhibit a mixture of papules, scabs and
scars up to several centimeters in length. These cysts also cover the corpse of
the wet-nurse Sitre-In, who was probably unrelated to the royal lineage. In
addition, Hatshepsut and her successor, Thutmose III (ca. 1,457-1,425 B.C.),
bear traces of the disease suggesting their skin healed after a period of time.
Recent DNA evidence suggests that Thutmose III might not be related to Thutmose
II. That Sitre-In and Thutmose III show evidence of this disease suggests the
disease was not hereditary but widely affected Thutmose II and his
court.